| Identifier | HPI045396 |
| Interaction | Ki-67 <==> p53 |
| Role & State | --- |
| Interaction Type | IT:0000203,stain; |
| Biological Process | BP:0040007,growth; |
| Biological Function | --- |
| Subcelluar Location | SCL:0000004,extracellular matrix; SCL:0000001,extracellular; |
| Detection Method | DM:0000089,array; |
| Reference | 18266006_2, The expression of Ki-67, caspase-3, p53, fragile histine triad (FHIT), maspin, extracellular matrix metalloproteinase inducer (EMMPRIN), vascular growth factor (VEGF), MUC-2, 4, 5AC and 6, CD44, E-cadherin, beta-catenin, and phosphorylated glycogen synthase kinase 3beta-ser9 (P-GSK3beta-ser9) was examined on tissue microarrays using immunohistochemistry.
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| Reference | 8863666_4, Expression of hepatocytic differentiation marker (HEP-PAR-reactive antigen), biliary differentiation markers (AE1-AE3, cytokeratin-19), proliferation markers (Ki-67, proliferating cell nuclear antigen), alpha-fetoprotein, p53, and transforming growth factor-alpha in the tumor tissue were assessed by immunohistochemistry.
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| Reference | 9772294_3, Ki-67 (MIB-1) immunostaining, argyrophilic nucleolar organizer regions and c-myc gene amplification were examined as parameters of proliferation, and p53 overexpression was examined in relation to clinicopathologic features and prognosis.
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